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Quidel Polyclonal Antiserum to Human C1 Inhibitor (C1 Esterase) A300|产品详情|进口榴莲视频免费观看采购网





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    订购信息
    上海榴莲视频下载地址入口生物科技公司
    Tel:400-968-7988    021-33779008
    Quidel Polyclonal Antiserum to Human C1 Inhibitor (C1 Esterase)
    品牌:Quidel
    货号:A300
    规格:2.0 mL
    货期:现货

    商品详情 参考文献 相关资料

    产品介绍

    品名

    Quidel Polyclonal Antiserum to Human C1 Inhibitor (C1 Esterase)

    货号

     A300

    规格

    2.0 mL 

     形式

    Whole Antiserum 

    防腐剂

     ≤ 0.1% Sodium Azide

    物种交叉反应

    Baboon, Dog, Rabbit

    储存

    2°C to 8°C  (≤ 30 days)

    For long-term storage (> 30 days), aliquot and store at ≤ –20°C. Avoid repeated freeze-thaw.

    使用说明

    For Research Use Only. Not for use in diagnostic procedures

    产品描述

    Highly purified human C1-inhibitor was isolated from normal serum and used to immunize goats. The C1-inhibitor polyclonal antisera was tested against normal human plasma by double immunodiffusion, one-dimensional immunoelectrophoresis, quantitative radial immunodiffusion, and quantitative rocket immunoelectrophoresis. The antiserum was determined to be monospecific for C1-inhibitor at varying concentrations.

    Quidel Polyclonal Antiserum to Human C1 Inhibitor (C1 Esterase)

    背景

    C1-inhibitor is a multispecific protease inhibitor present in human plasma and serum. Normal human serum shows a concentration of approximately 200 µg/mL. C1-inhibitor is a glycoprotein with an approximate molecular weight of 110 kD. This protein contains many glycosylation sites, so the molecular weight varies depending on the quantity of sites that are glycosylated

    应用

    Applications of the C1-inhibitor polyclonal antisera have been evalsuated by various research facilities, and include Western Blot 1 and Immunoblot. 2

    参考文献

    Schwab, J. et al. “Borrelia valaisiana Resist Complement-Mediated Killing Independently of the Recruitment of Immune Regulators and Inactivation of Complement Components.” PLoS ONE 8(1): e53659. 

    Yasojima, K., et al. “Complement Components, but Not Complement Inhibitors, are Upregulated in Atherosclerotic Plaques.” Arteriosesclerosis, Thrombosis, and Vascular Biology (2001): 1214-1219


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